The file for S1 Fig is incorrect in the published article. The correct S1 Fig can be viewed below.
Supporting information
S1 Fig. TNF secretion from L. pneumophila-infected BMDMs is not increased by LegC4 or loss of PA28αβ.
TNF WT or Psme1/2-/- BMDMs infected for (A) 8 h or (B) 24 h with L. pneumophila ΔflaA, ΔflaAΔlegC4 (pEV), ΔflaAΔlegC4 (plegC4), or the avirulent ΔdotA control at a multiplicity of infection of 10. Plasmid expression of legC4 was induced with 1 mM IPTG. Data shown are mean ± s.d. on samples in triplicates for a single experiment and are representative of results from three independent experiments. Asterisks denote statistical significance by two-way ANOVA (*P<0.05; **P<0.05). ns; not significant.
https://doi.org/10.1371/journal.ppat.1011604.s001
(TIF)
Reference
- 1. Ngwaga T, Chauhan D, Salberg AG, Shames SR (2023) Effector-mediated subversion of proteasome activator (PA)28αβ enhances host defense against Legionella pneumophila under inflammatory and oxidative stress conditions. PLoS Pathog 19(6): e1011473. https://doi.org/10.1371/journal.ppat.101147
Citation: Ngwaga T, Chauhan D, Salberg AG, Shames SR (2023) Correction: Effector-mediated subversion of proteasome activator (PA)28αβ enhances host defense against Legionella pneumophila under inflammatory and oxidative stress conditions. PLoS Pathog 19(8): e1011604. https://doi.org/10.1371/journal.ppat.1011604
Published: August 22, 2023
Copyright: © 2023 Ngwaga et al. This is an open access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.
